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Whole-exome sequencing of recurrent GBM suggests that these tumors arise from residual primary tumor cells, supporting a model of evolutionary adaptation to treatment

[,] In our patients case, the organism was sensitive to amikacin, gentamicin, ciprofloxacin, levofloxacin, and clarithromycin, and the patient was treated with a combination of piperacillin/tazobactam and ornidazole, which effectively improved the patients symptoms

The subsequent VDR gene silencing experiments revealed that while 1,25(OH)D continues to mitigate ferroptosis, its efficacy is diminished without VDR activation, highlighting the significance of VDR in its protective mechanism (Fig

WIREs Nanomed Nanobiotechnol 8, 579601

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